Published: 2 June 2016
Author(s): Mar Ariza, Natalia Cuenca, Marta Mauri, M. Angels Jurado, Maite Garolera
Issue: June 2016
Section: Letter to the Editor

Familial hypercholesterolemia (FH) is the most common disorder of lipid metabolism and can be caused by defects in different genes that code for proteins involved in hepatic clearance of low-density lipoprotein-cholesterol (LDL-C). FH subjects may develop premature atherosclerotic cardiovascular disease and they have a much higher risk of dying from a coronary event than do the general population [1]. In addition, FH patients appear to have an increased risk of cognitive impairment in old age [2,3].

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