Left ventricular (LV) thrombus is a known complication of acute myocardial infarction (AMI), with a reported incidence ranging from 0.7 to 4.3% in the era of primary percutaneous coronary intervention (PCI) [1, 2]. Previous studies have demonstrated that anterior ST segment elevation myocardial infarction (STEMI) and a left ventricular ejection fraction (LVEF) ≤ 40% are significant predictors of LV thrombus development [3]. On the other hand, non-STEMI (NSTEMI) represents a distinct pathophysiology and NSTEMI patients often undergo early, as opposed to immediate revascularisation as in STEMI patients [4].
