Amyloidosis is a multi-systemic disease resulting from deposition of misfolded proteins as insoluble fibrils in the interstitium of affected organs, subsequently leading to organ failure. Cardiac involvement in amyloidosis is common [1] and amyloid fibrils can deposit in several cardiac structures, including coronary arteries, ventricles, atria and heart valves [2,3]. Although more than 30 proteins can form amyloid fibrils, cardiac amyloidosis is typically attributable to misfolded transthyretin (ATTR) or immunoglobulin light chain (AL) aggregation [4].
