Published: 19 October 2020
Author(s): Leonard D. Browne, Fatima-Zahra Jaouimaa, Cathal Walsh, Fernando Perez-Ruiz, Paschal Richette, Kevin Burke, Austin G. Stack
Section: Original article

It is widely considered that serum uric acid (SUA), the final breakdown product of purine metabolism in humans, may represent a major cardiovascular risk factor and contribute to adverse health outcomes and mortality [1–6]. Elevated concentrations of SUA have been implicated in the development of hypertension, metabolic syndrome, type 2 diabetes, coronary artery disease, left ventricular hypertrophy, atrial fibrillation, myocardial infarction, stroke, heart failure and chronic kidney disease [7].


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